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- W3174010934 abstract "Recent work from our lab and other groups has indicated that inherent differences in microvascular architecture between mouse strains may be the underlying cause of variation in collateral expansion and capillary arterialization following a remodeling stimulus. We sought to examine these differences in C57BL/6 and BALB/c mice, strains with many and few/no collateral arteriolar connections, respectively, following ischemic ligation in the spinotrapezius muscle. In BALB/c mice, the percentage of actin-positive capillaries increased 190% seven days after ischemic ligation compared to controls, returning to near-control levels by 21 days. The total length of actin-positive vessels increased 52% seven days after ligation compared to controls, and was sustained out to 21 days. Ligated BALB/c muscles exhibit a 56% increase in small-arteriole length density seven days following treatment when compared to controls which was again sustained for 21 days. In C57BL/6 mice, ligated muscles had a 37% increase in large-arteriole length density 21 days after treatment compared to controls. No significant changes in capillary length density were observed following seven or 21 days of whole-body hypoxia in either strain. These results suggest that the presence or absence of arteriolar collateral connections influences the location and type of microvascular remodeling that occurs in the mouse spinotrapezius." @default.
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- W3174010934 date "2011-04-01" @default.
- W3174010934 modified "2023-10-16" @default.
- W3174010934 title "Collateral Expansion and Capillary Arterialization in the Spinotrapezius of C57BL/6, BALB/c and NG2 Knockout Mice" @default.
- W3174010934 doi "https://doi.org/10.1096/fasebj.25.1_supplement.1092.13" @default.
- W3174010934 hasPublicationYear "2011" @default.
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