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- W3184450707 endingPage "1019" @default.
- W3184450707 startingPage "1010" @default.
- W3184450707 abstract "Investigation of cancer as a cell-level disease has led to the development of cancer cell-directed therapies including cytotoxic T lymphocyte (CTL)-based immunotherapy; yet, many patients are refractory to these modalities of cancer treatment and acquired resistance frequently occurs. Of note, cancer environment controls the manifestation of cancerous cell phenotype. Helper T (Th) cells orchestrate immune defense responses targeting cancer cells as well as the tumor microenvironment. Recent studies have shown that in addition to interferon (IFN)-γ-producing Th1 cells, interleukin (IL)-4-producing Th2 cells function as potent anticancer effectors in part by promoting tumor stroma reconfiguration and tumor tissue repair. Such Th cell-mediated tissue-level immunity may be harnessed for novel modalities of cancer environment immunotherapy." @default.
- W3184450707 created "2021-08-02" @default.
- W3184450707 creator A5043641375 @default.
- W3184450707 creator A5064607675 @default.
- W3184450707 creator A5082780999 @default.
- W3184450707 date "2021-11-01" @default.
- W3184450707 modified "2023-10-15" @default.
- W3184450707 title "Immunity beyond cancer cells: perspective from tumor tissue" @default.
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- W3184450707 doi "https://doi.org/10.1016/j.trecan.2021.06.007" @default.
- W3184450707 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/8541902" @default.
- W3184450707 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/34305041" @default.
- W3184450707 hasPublicationYear "2021" @default.