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- W3187936685 endingPage "977" @default.
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- W3187936685 abstract "Epigenetic marks the genome by DNA methylation, histone modification or non-coding RNAs. Epigenetic marks instruct cells to respond reversibly to environmental cues and keep the specific gene expression stable throughout life. In this review, we concentrate on DNA methylation, the mechanism often associated with transgenerational persistence and for this reason frequently used in the clinic. A large study that included data from 10,000 blood samples detected 187 methylated sites associated with body mass index (BMI). The same study demonstrates that altered methylation results from obesity (OB). In another study the combined genetic and epigenetic analysis allowed us to understand the mechanism associating hepatic insulin resistance and non-alcoholic disease in Type 2 Diabetes (T2D) patients. The study underlines the therapeutic potential of epigenetic studies. We also account for seemingly contradictory results associated with epigenetics." @default.
- W3187936685 created "2021-08-16" @default.
- W3187936685 creator A5002936985 @default.
- W3187936685 creator A5026444785 @default.
- W3187936685 date "2021-08-08" @default.
- W3187936685 modified "2023-09-27" @default.
- W3187936685 title "Role of Epigenetics in Type 2 Diabetes and Obesity" @default.
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- W3187936685 doi "https://doi.org/10.3390/biomedicines9080977" @default.
- W3187936685 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/8393970" @default.
- W3187936685 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/34440181" @default.
- W3187936685 hasPublicationYear "2021" @default.