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• W3192545472 abstract "Recent progress has been made in understanding the roles and mechanisms of endoplasmic reticulum (ER) stress in the development and pathogenesis of diabetic nephropathy (DN). Hyperglycemia induces ER stress and apoptosis in renal cells. The induction of ER stress can be cytoprotective or cytotoxic. Experimental treatment of animals with ER stress inhibitors alleviated renal damage. Considering these findings, the normalization of ER stress by pharmacological agents is a promising approach to prevent or arrest DN progression. The current article reviews the mechanisms, roles, and therapeutic aspects of these findings." @default.
• W3192545472 created "2021-08-16" @default.
• W3192545472 creator A5037614030 @default.
• W3192545472 creator A5052069858 @default.
• W3192545472 creator A5079809318 @default.
• W3192545472 date "2021-08-02" @default.
• W3192545472 modified "2023-10-16" @default.
• W3192545472 title "Endoplasmic Reticulum Stress in Diabetic Nephrology: Regulation, Pathological Role, and Therapeutic Potential" @default.
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