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• W3195960892 abstract "Abstract Wound healing occurred with serial coordinated processes via coagulation-fibrinolysis, inflammation following to immune-activation, angiogenesis, granulation, and the final re-epithelization. Since the dermis forms critical physical and biological barriers, the repair system should be rapidly and accurately functioned to keep homeostasis in our body. The wound healing is impaired or dysregulated via an inappropriate microenvironment, which is easy to lead to several diseases, including fibrosis in multiple organs and psoriasis. Such a disease led to the dysregulation of several types of cells: immune cells, fibroblasts, mural cells, and endothelial cells. Moreover, recent progress in medical studies uncovers the significant concept. The calcium signaling, typically the following calcineurin-NFAT signaling, essentially regulates not only immune cell activations, but also various healing steps via coagulation, inflammation, and angiogenesis. In this review, we summarize the role of the NFAT activation pathway in wound healing and discuss its overall impact on future therapeutic ways." @default.
• W3195960892 created "2021-08-30" @default.
• W3195960892 creator A5012540837 @default.
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• W3195960892 date "2021-08-18" @default.
• W3195960892 modified "2023-10-16" @default.
• W3195960892 title "Calcineurin-nuclear factor for activated T cells (NFAT) signaling in pathophysiology of wound healing" @default.
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• W3195960892 doi "https://doi.org/10.1186/s41232-021-00176-5" @default.
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