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- W3199065741 endingPage "2517" @default.
- W3199065741 startingPage "2517" @default.
- W3199065741 abstract "Autophagy has been described as harboring a dual role in cancer development and therapy. Depending on the context, it can exert either pro-survival or pro-death functions. Here, we review what is known about autophagy in crizotinib-treated ALK+ ALCL. We first present our main findings on the role and regulation of autophagy in these cells. Then, we provide literature-driven hypotheses that could explain mechanistically the pro-survival properties of autophagy in crizotinib-treated bulk and stem-like ALK+ ALCL cells. Finally, we discuss how the potentiation of autophagy, which occurs with combined therapies (ALK and BCL2 or ALK and RAF1 co-inhibition), could convert it from a survival mechanism to a pro-death process." @default.
- W3199065741 created "2021-09-27" @default.
- W3199065741 creator A5060499506 @default.
- W3199065741 creator A5061642237 @default.
- W3199065741 creator A5077633685 @default.
- W3199065741 date "2021-09-23" @default.
- W3199065741 modified "2023-09-30" @default.
- W3199065741 title "The Dual Role of Autophagy in Crizotinib-Treated ALK+ ALCL: From the Lymphoma Cells Drug Resistance to Their Demise" @default.
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