Matches in SemOpenAlex for { <https://semopenalex.org/work/W3200471691> ?p ?o ?g. }
- W3200471691 endingPage "1468" @default.
- W3200471691 startingPage "1468" @default.
- W3200471691 abstract "Huntington's disease (HD) is caused by expansion of polyglutamine repeats in the protein huntingtin, which affects the corpus striatum of the brain. The polyglutamine repeats in mutant huntingtin cause its aggregation and elicit toxicity by affecting several cellular processes, which include dysregulated organellar stress responses. The Golgi apparatus not only plays key roles in the transport, processing, and targeting of proteins, but also functions as a sensor of stress, signaling through the Golgi stress response. Unlike the endoplasmic reticulum (ER) stress response, the Golgi stress response is relatively unexplored. This review focuses on the molecular mechanisms underlying the Golgi stress response and its intersection with cysteine metabolism in HD." @default.
- W3200471691 created "2021-09-27" @default.
- W3200471691 creator A5006367625 @default.
- W3200471691 date "2021-09-15" @default.
- W3200471691 modified "2023-10-14" @default.
- W3200471691 title "Signaling Overlap between the Golgi Stress Response and Cysteine Metabolism in Huntington’s Disease" @default.
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