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- W3200769106 endingPage "4190" @default.
- W3200769106 startingPage "4190" @default.
- W3200769106 abstract "Endothelial cells that cover the lumen of all blood vessels have the inherent capacity to express both pro and anticoagulant molecules. However, under normal physiological condition, they generally function to maintain a non-thrombogenic surface for unobstructed blood flow. In response to injury, certain stimuli, or as a result of dysfunction, endothelial cells release a highly adhesive procoagulant protein, von Willebrand factor (VWF), which plays a central role in formation of platelet aggregates and thrombus generation. Since VWF expression is highly restricted to endothelial cells, regulation of its levels is among the most important functions of endothelial cells for maintaining hemostasis. However, with aging, there is a significant increase in VWF levels, which is concomitant with a significant rise in thrombotic events. It is not yet clear why and how aging results in increased VWF levels. In this review, we have aimed to discuss the age-related increase in VWF, its potential mechanisms, and associated coagulopathies as probable consequences." @default.
- W3200769106 created "2021-09-27" @default.
- W3200769106 creator A5006557500 @default.
- W3200769106 creator A5024271944 @default.
- W3200769106 creator A5070411706 @default.
- W3200769106 date "2021-09-16" @default.
- W3200769106 modified "2023-10-16" @default.
- W3200769106 title "Age-Associated Increase in Thrombogenicity and Its Correlation with von Willebrand Factor" @default.
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