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- W3201044894 abstract "AKT is the central phosphoinositide 3-kinase (PI3K) signaling effector, however, PIK3CA (p110α subunit of PI3Kα)-mutant estrogen receptor-positive (ER+) breast cancers exhibit minimal AKT activation and the downstream signaling is poorly characterized. We discovered that a subset of PIK3CA-mutant ER+ breast cancers exhibit increased inositol polyphosphate 4-phosphatase type II (INPP4B) expression, which promotes late endosome formation and glycogen synthase kinase 3 beta (GSK3β) trafficking, leading to enhanced Wingless-related integration site (WNT)/catenin beta 1 (β-catenin) activation." @default.
- W3201044894 created "2021-09-27" @default.
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- W3201044894 date "2021-07-04" @default.
- W3201044894 modified "2023-09-25" @default.
- W3201044894 title "A late endosome signaling hub that couples PI3Kα and WNT/β-catenin signaling in breast cancer" @default.
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- W3201044894 doi "https://doi.org/10.1080/23723556.2021.1954470" @default.
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