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- W3204179210 endingPage "102" @default.
- W3204179210 startingPage "89" @default.
- W3204179210 abstract "The ongoing coronavirus disease-2019 (COVID-19) pandemic is caused by severe acute respiratory syndrome coronavirus-2. COVID-19 severity is related to the cytokine storm phenomenon, which is amplified by pro-inflammatory cytokines and chemokines; it may cause extensive pulmonary damage. Among these cytokines, interleukin (IL)-17, produced mainly by T helper 17 (Th17) cells, is responsible for the immunopathological responses present in acute respiratory distress syndrome. This review discusses the roles of IL-17 and Th17 responses in the pathophysiology of COVID-19. Dysregulated Th17-responses, linked to various risk factors, may contribute to pathological inflammation through the amplification of multiple inflammatory cytokines and chemokines, as well as augmentation of neutrophil infiltration in the lungs of severe COVID-19 patients. A more detailed understanding of the roles of Th17 responses, as well as the mechanisms underlying altered IL-17 production and signaling, may improve therapeutic strategies for severe or critically ill COVID-19 patients by targeting the IL-17 pathway." @default.
- W3204179210 created "2021-10-11" @default.
- W3204179210 creator A5018204590 @default.
- W3204179210 creator A5027254842 @default.
- W3204179210 creator A5047657591 @default.
- W3204179210 creator A5089677398 @default.
- W3204179210 date "2021-09-01" @default.
- W3204179210 modified "2023-10-18" @default.
- W3204179210 title "Roles of Interleukin-17 and Th17 Responses in COVID-19" @default.
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