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- W3204604593 abstract "Pemphigoid diseases (PD) comprise a group of autoimmune disorders clinically characterized by (muco)-cutaneous inflammation and subepidermal blistering. PD are caused by autoantibodies targeting structural proteins of the dermal-epidermal junction. Skin-bound immune complexes trigger recruitment of myeloid cells, engagement of Fcγ receptors, and activation of specific kinases. Selective blockade of PI3K can alleviate disease manifestation in mouse models of autoimmune diseases. Here, we evaluated the treatment efficacy of parsaclisib, a selective PI3KF064 inhibitor, in in-vitro and in-vivo models of epidermolysis bullosa acquisita (EBA) and mucous membrane pemphigoid (MMP)." @default.
- W3204604593 created "2021-10-11" @default.
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- W3204604593 date "2021-10-01" @default.
- W3204604593 modified "2023-09-26" @default.
- W3204604593 title "018 Cutaneous kinase activity correlates with treatment outcomes following PI3K delta inhibition in mice with experimental pemphigoid diseases" @default.
- W3204604593 doi "https://doi.org/10.1016/j.jid.2021.08.019" @default.
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