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- W3207259968 abstract "Recently, the medications used for the severe form of the coronavirus disease-19 (COVID-19) therapy are of particular interest. In this sense, it has been supposed that anti-VEGF compounds would be good candidates in the face of “cytokine storm” and intussuscepted angiogenesis due to having an appreciable anti-inflammatory effect. Therefore, they can be subjected to therapeutic protocols to manage acute respiratory distress syndrome (ARDS). Since the compelling evidence emphasized that VEGFs contribute to the inflammatory process and play a mainstay role in disease pathogenesis, in this review, we aimed to highlight the VEGF's plausible participation in the cytokine storm exacerbation in COVID-19. Next, the recent clinical advances regarding the anti-VEGF medications, including humanized monoclonal antibody, immunosuppressant, a tyrosine kinase inhibitor, and a cytokine inhibitor, have been addressed in the setting of COVID-19 treatment in critically ill patients. Together, retrieving the increased level of VEGF subsets, as well as antagonizing VEGF related receptors, could be helpful for the treatment of COVID-19, especially in those suffering from ARDS." @default.
- W3207259968 created "2021-10-25" @default.
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- W3207259968 date "2021-12-01" @default.
- W3207259968 modified "2023-09-26" @default.
- W3207259968 title "Anti-VEGF agents: As appealing targets in the setting of COVID-19 treatment in critically ill patients" @default.
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- W3207259968 doi "https://doi.org/10.1016/j.intimp.2021.108257" @default.
- W3207259968 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/8519896" @default.
- W3207259968 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/34673299" @default.
- W3207259968 hasPublicationYear "2021" @default.
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