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• W3207423903 endingPage "11159" @default.
• W3207423903 startingPage "11159" @default.
• W3207423903 abstract "Non-ionizing radiation is commonly used in the clinical setting, despite its known ability to trigger oxidative stress and apoptosis, which can lead to damage and cell death. Although induction of cell death is typically considered harmful, apoptosis can also be beneficial in the right context. For example, cell death can serve as the signal for new tissue growth, such as in apoptosis-induced proliferation. Recent data has shown that exposure to non-ionizing radiation (such as weak static magnetic fields, weak radiofrequency magnetic fields, and weak electromagnetic fields) is able to modulate proliferation, both in cell culture and in living organisms (for example during tissue regeneration). This occurs via in vivo changes in the levels of reactive oxygen species (ROS), which are canonical activators of apoptosis. This review will describe the literature that highlights the tantalizing possibility that non-ionizing radiation could be used to manipulate apoptosis-induced proliferation to either promote growth (for regenerative medicine) or inhibit it (for cancer therapies). However, as uncontrolled growth can lead to tumorigenesis, much more research into this exciting and developing area is needed in order to realize its promise." @default.
• W3207423903 created "2021-10-25" @default.
• W3207423903 creator A5041636768 @default.
• W3207423903 creator A5047881447 @default.
• W3207423903 creator A5048756641 @default.
• W3207423903 date "2021-10-16" @default.
• W3207423903 modified "2023-10-10" @default.
• W3207423903 title "An Open Question: Is Non-Ionizing Radiation a Tool for Controlling Apoptosis-Induced Proliferation?" @default.
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• W3207423903 doi "https://doi.org/10.3390/ijms222011159" @default.
• W3207423903 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/8537877" @default.
• W3207423903 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/34681819" @default.

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