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- W3208049697 abstract "To date, nearly 100 autoimmune diseases have been an area of focus, and these diseases bring health challenges to approximately 5% of the population worldwide. As a type of disease caused by tolerance breakdown, both environmental and genetic risk factors contribute to autoimmune disease development. However, in most cases, there are still gaps in our understanding of disease pathogenesis, diagnosis, and treatment. Therefore, more detailed knowledge of disease pathogenesis and potential therapies is indispensable. DNA methylation, which does not affect the DNA sequence, is one of the key epigenetic silencing mechanisms and has been indicated to play a key role in gene expression regulation and to participate in the development of certain autoimmune diseases. Potential epigenetic regulation via DNA methylation has garnered more attention as a disease biomarker in recent years. In this review, we clarify the basic function and distribution of DNA methylation, evaluate its effects on gene expression and discuss related key enzymes. In addition, we summarize recent aberrant DNA methylation modifications identified in the most important cell types related to several autoimmune diseases and then provide potential directions for better diagnosing and monitoring disease progression driven by epigenetic control, which may broaden our understanding and contribute to further epigenetic research in autoimmune diseases." @default.
- W3208049697 created "2021-11-08" @default.
- W3208049697 creator A5005244980 @default.
- W3208049697 creator A5022677475 @default.
- W3208049697 creator A5023387174 @default.
- W3208049697 creator A5058090939 @default.
- W3208049697 creator A5074571072 @default.
- W3208049697 creator A5079650987 @default.
- W3208049697 creator A5079738340 @default.
- W3208049697 date "2021-11-01" @default.
- W3208049697 modified "2023-10-03" @default.
- W3208049697 title "Insights Into the Role of DNA Methylation in Immune Cell Development and Autoimmune Disease" @default.
- W3208049697 cites W1540298277 @default.
- W3208049697 cites W1567305408 @default.
- W3208049697 cites W1607528816 @default.
- W3208049697 cites W1674079630 @default.
- W3208049697 cites W1874574653 @default.
- W3208049697 cites W1948252677 @default.
- W3208049697 cites W1964249723 @default.
- W3208049697 cites W1970464961 @default.
- W3208049697 cites W1977165060 @default.
- W3208049697 cites W1979892065 @default.
- W3208049697 cites W1983019708 @default.
- W3208049697 cites W1989354231 @default.
- W3208049697 cites W1998137132 @default.
- W3208049697 cites W2004172747 @default.
- W3208049697 cites W2011730338 @default.
- W3208049697 cites W2014634821 @default.
- W3208049697 cites W2015888582 @default.
- W3208049697 cites W2018276288 @default.
- W3208049697 cites W2020658841 @default.
- W3208049697 cites W2020775355 @default.
- W3208049697 cites W2022081039 @default.
- W3208049697 cites W2023258555 @default.
- W3208049697 cites W2026336857 @default.
- W3208049697 cites W2030761948 @default.
- W3208049697 cites W2032917789 @default.
- W3208049697 cites W2037088576 @default.
- W3208049697 cites W2040850093 @default.
- W3208049697 cites W2048132100 @default.
- W3208049697 cites W2049165141 @default.
- W3208049697 cites W2049977668 @default.
- W3208049697 cites W2053856108 @default.
- W3208049697 cites W2054281806 @default.
- W3208049697 cites W2064943181 @default.
- W3208049697 cites W2067036337 @default.
- W3208049697 cites W2068511191 @default.
- W3208049697 cites W2072959464 @default.
- W3208049697 cites W2075276275 @default.
- W3208049697 cites W2079562528 @default.
- W3208049697 cites W2080322382 @default.
- W3208049697 cites W2091503140 @default.
- W3208049697 cites W2094011807 @default.
- W3208049697 cites W2104960097 @default.
- W3208049697 cites W2110480756 @default.
- W3208049697 cites W2114912562 @default.
- W3208049697 cites W2119443108 @default.
- W3208049697 cites W2121417064 @default.
- W3208049697 cites W2124049725 @default.
- W3208049697 cites W2125503507 @default.
- W3208049697 cites W2130014053 @default.
- W3208049697 cites W2133300386 @default.
- W3208049697 cites W2138699372 @default.
- W3208049697 cites W2139285516 @default.
- W3208049697 cites W2139891717 @default.
- W3208049697 cites W2146152969 @default.
- W3208049697 cites W2148521032 @default.
- W3208049697 cites W2148993123 @default.
- W3208049697 cites W2150250444 @default.
- W3208049697 cites W2151835486 @default.
- W3208049697 cites W2152656267 @default.
- W3208049697 cites W2153886009 @default.
- W3208049697 cites W2161250377 @default.
- W3208049697 cites W2168069435 @default.
- W3208049697 cites W2209371252 @default.
- W3208049697 cites W2225123924 @default.
- W3208049697 cites W2226995712 @default.
- W3208049697 cites W2253043407 @default.
- W3208049697 cites W2271166222 @default.
- W3208049697 cites W2285456194 @default.
- W3208049697 cites W2286983359 @default.
- W3208049697 cites W2335122682 @default.
- W3208049697 cites W2406303635 @default.
- W3208049697 cites W2414449891 @default.
- W3208049697 cites W2515561881 @default.
- W3208049697 cites W2527889668 @default.
- W3208049697 cites W2553358001 @default.
- W3208049697 cites W2557683813 @default.
- W3208049697 cites W2606785272 @default.
- W3208049697 cites W2615078726 @default.
- W3208049697 cites W2732179863 @default.
- W3208049697 cites W2748019834 @default.
- W3208049697 cites W2766643978 @default.
- W3208049697 cites W2774214122 @default.
- W3208049697 cites W2782832103 @default.
- W3208049697 cites W2786704362 @default.
- W3208049697 cites W2787214556 @default.
- W3208049697 cites W2804716665 @default.
- W3208049697 cites W2881685221 @default.
- W3208049697 cites W2905710996 @default.