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- W3208802014 abstract "Objective: Previous studies have found a hot HPV integration site, CCDC106, in a 61 cervical intraepithelial neoplasia (CIN) cohort, with high expression of CCDC106 caused by HPV-CCDC106 integration in the Hi-C dataset. We then found that all the transcription of HPV16 E6 was the form of E6 splice (E6*I) in these carcinoma samples. HPV16 E6 promoted the development of cervical cancer, but the effect of E6 splices, especially E6*I, on cervical cancer was known to be controversial. The effect of high expression of CCDC106 accompanying the expression of E6*I in cervical carcinogenesis is not clear." @default.
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- W3208802014 date "2020-10-01" @default.
- W3208802014 modified "2023-09-23" @default.
- W3208802014 title "High expression of CCDC106 promotes cervical cancer cell proliferation and migration by p53 degradation despite the HPV16 E6 spliced status" @default.
- W3208802014 doi "https://doi.org/10.1016/j.ygyno.2020.05.289" @default.
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