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- W320904362 abstract "EH was complex genetic diseases, which was the result caused by the affect of a class of multiple genes and environmental factors. The impact of changes in blood pressure accounted for 30%-50% [2]. Since 1992, the first report by Jeune Maitre reported that angiotensinogen (angiotensinogen, AGT) gene polymorphisms was realted EH gene creating a gene polymorphism EH associated studies which set a precedent. Among the many candidate genes for EH, centering on the renin-angiotensin-aldosterone system, sympathetic nervous system, hypothalamus-pituitary axis, endothelin, natriuretic peptide, kallikrein-kinin system at least 150 candidates EH genes have been extensively studied [3]. Among the many candidate genes endothelial nitric oxide synthase (endothelial nitric oxide synthase, eNOS) gene was increased attention due to the to the important role devoted to the regulation of blood pressure. The paper was a summary for the relevance of ENOS polymorphisms and EH." @default.
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- W320904362 date "2009-01-01" @default.
- W320904362 modified "2023-09-24" @default.
- W320904362 title "Research progress of relativity between eNOS polymorphic and essential hypertension." @default.
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