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- W3211061444 endingPage "108856" @default.
- W3211061444 startingPage "108856" @default.
- W3211061444 abstract "Alcohol dependence is characterized by a shift in motivation to consume alcohol from positive reinforcement (i.e., increased likelihood of future alcohol drinking based on its rewarding effects) to negative reinforcement (i.e., increased likelihood of future alcohol drinking based on alcohol-induced reductions in negative affective symptoms, including but not limited to those experienced during alcohol withdrawal). The neural adaptations that occur during this transition are not entirely understood. Mesolimbic reinforcement circuitry (i.e., ventral tegmental area [VTA] neurons) is activated during early stages of alcohol use, and may be involved in the recruitment of brain stress circuitry (i.e., extended amygdala) during the transition to alcohol dependence, after chronic periods of high-dose alcohol exposure. Here, we review the literature regarding the role of canonical brain reinforcement (VTA) and brain stress (extended amygdala) systems, and the connections between them, in acute, sub-chronic, and chronic alcohol response. Particular emphasis is placed on preclinical models of alcohol use. This article is part of the special Issue on ‘Neurocircuitry Modulating Drug and Alcohol Abuse'." @default.
- W3211061444 created "2021-11-08" @default.
- W3211061444 creator A5003204165 @default.
- W3211061444 creator A5043734937 @default.
- W3211061444 date "2022-01-01" @default.
- W3211061444 modified "2023-10-09" @default.
- W3211061444 title "Reciprocal midbrain-extended amygdala circuit activity in preclinical models of alcohol use and misuse" @default.
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