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- W3211125014 abstract "Adolescent polycystic ovary syndrome (PCOS) is most often the outcome of a mismatch between an energy-sparing (epi)genetic background and a relatively energy-rich environment. This mismatch results in a sustained requirement to store more fat than is safely feasible in subcutaneous adipose tissue, and the excess of fat ends up being stored in ectopic depots, notably in the liver and viscera (= hepato-visceral or central fat). In adolescent girls with PCOS, prolonged treatment with an estro-progestagen contraceptive reduces the androgen excess but not the underpinning PCOS drive (exerted by ectopic fat excess and insulin resistance) so that, upon discontinuation of such prolonged treatment, the young women rebound swiftly into androgen excess and oligo-anovulation, thus into adult PCOS." @default.
- W3211125014 created "2021-11-08" @default.
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- W3211125014 date "2021-01-01" @default.
- W3211125014 modified "2023-09-25" @default.
- W3211125014 title "Toward Adolescent Prevention of Adult Anovulation in Polycystic Ovary Syndrome" @default.
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- W3211125014 doi "https://doi.org/10.1007/978-3-030-63650-0_3" @default.
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