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- W3214621185 abstract "Chronic kidney disease (CKD) is associated with increased risk of cardiovascular (CV) events, and the disease burden is rising rapidly. An important contributor to CV events and CKD progression is high blood pressure (BP). The main mechanisms of hypertension in early and advanced CKD are renin-angiotensin system activation and volume overload, respectively. Sodium retention is well known as a factor for high BP in CKD. However, a BP increase in response to total body sodium or volume overload can be limited by neurohormonal modulation. Recent clinical trial data favoring intensive BP lowering in CKD imply that the balance between volume and neurohormonal control could be revisited with respect to the safety and efficacy of strict volume control when using antihypertensive medications. In hemodialysis patients, the role of more liberal use of antihypertensive medications with the concept of functional dry weight for intensive BP control must be studied." @default.
- W3214621185 created "2021-11-22" @default.
- W3214621185 creator A5051877787 @default.
- W3214621185 creator A5079349620 @default.
- W3214621185 date "2021-12-31" @default.
- W3214621185 modified "2023-10-12" @default.
- W3214621185 title "The roles of sodium and volume overload on hypertension in chronic kidney disease" @default.
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- W3214621185 doi "https://doi.org/10.23876/j.krcp.21.800" @default.
- W3214621185 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/34922428" @default.
- W3214621185 hasPublicationYear "2021" @default.
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