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• W3215724636 abstract "Pseudomonas aeruginosa and Staphylococcus aureus are both opportunistic pathogens that are frequently associated with chronic lung infections. While bacterial virulence determinants are critical in initiating infection, the metabolic flexibility of these bacteria promotes their persistence in the airway. Upon infection, these pathogens induce host immunometabolic reprogramming, resulting in an airway milieu replete with immune-signaling metabolites. These metabolites are often toxic to the bacteria and create a steep selection pressure for the emergence of bacterial isolates adapted for long-term survival in the inflamed lung. In this review, we discuss the main differences in the host immunometabolic response to P. aeruginosa and S. aureus, as well as how these pathogens alter their own metabolism to adapt to airway metabolites and cause persistent lung infections." @default.
• W3215724636 created "2021-12-06" @default.
• W3215724636 creator A5070933756 @default.
• W3215724636 creator A5078115460 @default.
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• W3215724636 date "2021-11-25" @default.
• W3215724636 modified "2023-10-12" @default.
• W3215724636 title "Immunometabolites Drive Bacterial Adaptation to the Airway" @default.
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• W3215724636 doi "https://doi.org/10.3389/fimmu.2021.790574" @default.
• W3215724636 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/34899759" @default.
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