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- W3215863333 abstract "Chronic stress is common among cancer patients due to the psychological, operative, or pharmaceutical stressors at the time of diagnosis or during the treatment of cancers. The continuous activations of the hypothalamic-pituitary-adrenal (HPA) axis and the sympathetic nervous system (SNS), as results of chronic stress, have been demonstrated to take part in several cancer-promoting processes, such as tumorigenesis, progression, metastasis, and multi-drug resistance, by altering the tumor microenvironment (TME). Stressed TME is generally characterized by the increased proportion of cancer-promoting cells and cytokines, the reduction and malfunction of immune-supportive cells and cytokines, augmented angiogenesis, enhanced epithelial-mesenchymal transition, and damaged extracellular matrix. For the negative effects that these alterations can cause in terms of the efficacies of anti-cancer treatments and prognosis of patients, supplementary pharmacological or psychotherapeutic strategies targeting HPA, SNS, or psychological stress may be effective in improving the prognosis of cancer patients. Here, we review the characteristics and mechanisms of TME alterations under chronic stress, their influences on anti-cancer therapies, and accessory interventions and therapies for stressed cancer patients." @default.
- W3215863333 created "2021-12-06" @default.
- W3215863333 creator A5000295654 @default.
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- W3215863333 creator A5008659449 @default.
- W3215863333 creator A5009198836 @default.
- W3215863333 creator A5049181967 @default.
- W3215863333 creator A5064509097 @default.
- W3215863333 creator A5078121940 @default.
- W3215863333 creator A5085633511 @default.
- W3215863333 date "2021-11-19" @default.
- W3215863333 modified "2023-10-18" @default.
- W3215863333 title "Chronic Stress: Impacts on Tumor Microenvironment and Implications for Anti-Cancer Treatments" @default.
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