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• W3216072110 startingPage "48" @default.
• W3216072110 abstract "Hyperglycemia, which occurs under the diabetic conditions, induces serious diabetic complications. Diabetic encephalopathy has been defined as one of the major complications of diabetes, and is characterized by neurochemical and neurodegenerative changes. However, little is known about the effect of long-term exposure to high glucose on neuronal cells. In the present study, we showed that exposure to glutamate (100 mM) for 7 days induced toxicity in primary cortical neurons using the MTT assay. Additionally, high glucose increased the sensitivity of AMPA- or NMDA-induced neurotoxicity, and decreased extracellular glutamate levels in primary cortical neurons. In Western blot analyses, the protein levels of the GluA1 and GluA2 subunits of the AMPA receptor as well as synaptophysin in neurons treated with high glucose were significantly increased compared with the control (25 mM glucose). Therefore, long-term exposure to high glucose induced neuronal death through the disruption of glutamate homeostasis." @default.
• W3216072110 created "2021-12-06" @default.
• W3216072110 creator A5030814151 @default.
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• W3216072110 date "2022-01-01" @default.
• W3216072110 modified "2023-09-30" @default.
• W3216072110 title "Long-term exposure to high glucose induces changes in the expression of AMPA receptor subunits and glutamate transmission in primary cultured cortical neurons" @default.
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• W3216072110 doi "https://doi.org/10.1016/j.bbrc.2021.11.108" @default.
• W3216072110 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/34891041" @default.
• W3216072110 hasPublicationYear "2022" @default.
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