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- W3217461162 abstract "Among the long-term consequences of sepsis (also termed post-sepsis syndrome) the increased risk of unexplained cardiovascular complications, such as myocardial infarction, acute heart failure or stroke, is one of the emerging specific health concerns. The vascular accelerated ageing also named premature senescence is a potential mechanism contributing to atherothrombosis, consequently leading to cardiovascular events. Indeed, vascular senescence-associated major adverse cardiovascular events (MACE) are a potential feature in sepsis survivors and of the elderly at cardiovascular risk. In these patients, accelerated vascular senescence could be one of the potential facilitating mechanisms. This review will focus on premature senescence in sepsis regardless of age. It will highlight and refine the potential relationships between sepsis and accelerated vascular senescence. In particular, key cellular mechanisms contributing to cardiovascular events in post-sepsis syndrome will be highlighted, and potential therapeutic strategies to reduce the cardiovascular risk will be further discussed." @default.
- W3217461162 created "2021-12-06" @default.
- W3217461162 creator A5031384139 @default.
- W3217461162 creator A5038554072 @default.
- W3217461162 creator A5068617049 @default.
- W3217461162 creator A5069652712 @default.
- W3217461162 date "2021-12-01" @default.
- W3217461162 modified "2023-10-17" @default.
- W3217461162 title "Long-term cardiovascular complications following sepsis: is senescence the missing link?" @default.
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