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- W3217583022 endingPage "12687" @default.
- W3217583022 startingPage "12687" @default.
- W3217583022 abstract "Chronic alcohol consumption and alcohol-associated liver disease (ALD) represent a major public health problem worldwide. Only a minority of patients with an alcohol-use disorder (AUD) develop severe forms of liver disease (e.g., steatohepatitis and fibrosis) and finally progress to the more advanced stages of ALD, such as severe alcohol-associated hepatitis and decompensated cirrhosis. Emerging evidence suggests that gut barrier dysfunction is multifactorial, implicating microbiota changes, alterations in the intestinal epithelium, and immune dysfunction. This failing gut barrier ultimately allows microbial antigens, microbes, and metabolites to translocate to the liver and into systemic circulation. Subsequent activation of immune and inflammatory responses contributes to liver disease progression. Here we review the literature about the disturbance of the different host defense mechanisms linked to gut barrier dysfunction, increased microbial translocation, and impairment of liver and systemic inflammatory responses in the different stages of ALD." @default.
- W3217583022 created "2021-12-06" @default.
- W3217583022 creator A5001918029 @default.
- W3217583022 creator A5012576740 @default.
- W3217583022 creator A5052593818 @default.
- W3217583022 creator A5073591316 @default.
- W3217583022 date "2021-11-24" @default.
- W3217583022 modified "2023-10-02" @default.
- W3217583022 title "Host Factors in Dysregulation of the Gut Barrier Function during Alcohol-Associated Liver Disease" @default.
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