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• W3217736618 abstract "Hundreds of millions of people worldwide suffer from peripheral nerve damage resulting from car accidents, falls, industrial accidents, residential accidents, and wars. The purpose of our study was to further investigate the effects of Wallerian degeneration (WD) after rat sciatic nerve injury and to screen for critical long noncoding RNAs (lncRNAs) in WD. We found H19 to be essential for nerve degeneration and regeneration and to be highly expressed in the sciatic nerves of rats with WD. lncRNA H19 potentially impaired the recovery of sciatic nerve function in rats. H19 was mainly localized in the cytoplasm of Schwann cells (SCs) and promoted their migration. H19 promoted the apoptosis of dorsal root ganglion (DRG) neurons and slowed the growth of DRG axons. The lncRNA H19 may play a role in WD through the Wnt/β-catenin signaling pathway and is coexpressed with a variety of crucial mRNAs during WD. These data provide further insight into the molecular mechanisms of WD." @default.
• W3217736618 created "2021-12-06" @default.
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• W3217736618 date "2021-12-10" @default.
• W3217736618 modified "2023-10-15" @default.
• W3217736618 title "Effect of lncRNA H19 on nerve degeneration and regeneration after sciatic nerve injury in rats" @default.
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• W3217736618 doi "https://doi.org/10.1002/dneu.22861" @default.
• W3217736618 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/34818452" @default.
• W3217736618 hasPublicationYear "2021" @default.
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