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- W324790881 abstract "In isolated blood vessels, norepinephrine, whether released from adrenergic nerve terminals or added exogenously, interacts with alpha-adrenergic receptors on the vascular smooth muscle cells and causes their contraction. Depending on the anatomical origin of the studied vessel, the contractile responses to norepinephrine, as those to other vasoactive substances, are affected differently by factors such as the inhibition of calcium influx, local cooling, or anoxia; such differences are particularly obvious when arterial and venous smooth muscle are compared. In the blood vessel wall, norepinephrine not only interacts with postjunctional but also with prejunctional alpha-adrenoceptors that reduce the exocytotic release of the adrenergic neurotransmitter. Evidence suggests that the postjunctionol alpha-adrenoceptors of the blood vessel wall are not a homogenous group in terms of their pharmacological properties. The chapter compares the pharmacological characteristics of the postjunctional alpha-adrenoceptors in canine arteries and veins. When it appears that differences in alpha-adrenoceptor population exist between pre- and post-capillary vessels, an attempt is made to correlate them with differences in the handling of activator Ca2+ known to exist between arterial and venous smooth muscle." @default.
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- W324790881 date "1981-01-01" @default.
- W324790881 modified "2023-09-26" @default.
- W324790881 title "UNEVEN DISTRIBUTION OF POSTJUNCTIONAL ALPHA-ADRENERGIC RECEPTORS AND HANDLING OF Ca2+ IN CANINE VASCULAR SMOOTH MUSCLE" @default.
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- W324790881 doi "https://doi.org/10.1016/b978-0-08-026821-7.50037-3" @default.
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