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- W326540360 abstract "Changes in extracellular potassium concentration can significantly impact electrophysiological properties of the myocardial cell membrane. Hypokalemia can hyperpolarize the resting potential, increase the rate of spontaneous depolarization during phase four, increase the rate of rise of the membrane potential during the upstroke of the action potential, prolong the plateau phase of depolarization, delay repolarization, and increase the duration of the refractory period. These changes increase automaticity of the myocardial cells and increase the rate of conduction of impulses through the myocardium. The mechanism by which arrhythmias are related to hypokalemia is incompletely understood, although it is likely that the enhancement of automaticity, increased conduction velocity and the delayed ventricular repolarization work synergistically to predispose the myocardium to reentrant forms of arrhythmias. Taken together, clinical studies support the existence of an strong, inverse relationship between plasma potassium concentration and frequency of ventricular tachycardia and fibrillation after myocardial infarction." @default.
- W326540360 created "2016-06-24" @default.
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- W326540360 date "2001-01-01" @default.
- W326540360 modified "2023-09-25" @default.
- W326540360 title "Arrhythmogenic Significance of Hypokalemia" @default.
- W326540360 doi "https://doi.org/10.1007/978-1-4615-1443-5_10" @default.
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