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- W33018422 endingPage "121" @default.
- W33018422 startingPage "87" @default.
- W33018422 abstract "Epilepsies are defined as disorders of neuronal excitability characterized by periodic and unpredictable occurrences of seizures, while “seizures” are defined as transient changes of behaviour due to the disordered, synchronous, and rhythmic firing of populations of central nervous system neurons (McNamara 1994). These disorders of neuronal excitability can remain local or spread to other sites or engage all cortical regions simultaneously. Epileptogenesis could be defined as the molecular or cellular events producing the transient, disordered firing of a subpopulation of neurons in a key region of the brain, resulting in periodic seizures. A widely held view is that these seizures are caused by an abnormality in the major neurotransmitter systems of the brain such as excessive activity of the excitatory transmitters or impaired activity of the inhibitory transmitters, or a combination of both. However, the marked heterogeneity of syndromes (up to 50) diagnosed as epilepsy makes it most unlikely that there is a singular biochemical disorder that results in epileptogenesis. Furthermore, seizures set in motion a cascade of complex molecular and genomic changes, including changes in gene expression, sprouting of fibres, establishment of new synaptic contacts, alterations in expression of transmitters, modification of receptor expression, etc., that may contribute to the abnormally increased neuronal excitability and be responsible for a seizure-induced neuronal lesion." @default.
- W33018422 created "2016-06-24" @default.
- W33018422 creator A5017545603 @default.
- W33018422 date "1999-01-01" @default.
- W33018422 modified "2023-09-27" @default.
- W33018422 title "Epileptogenesis: Biochemical Aspects" @default.
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