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- W33601929 abstract "The increased independent risk for premature cardiovascular mortality related to left ventricular hypertrophy (LVH) may be attributed to a number of underlying pathophysiological alterations(1). Among the postulated mechanisms are: an intrinsic pathological defect inherent with the hypertrophied myocardium, per se; impaired ventricular pumping ability that may exacerbate decompensation; co-existent diseases of the left ventricle (e.g., occlusive epicardial artery atherosclerosis, exogenous obesity, diabetes mellitus); increased myocardial irritability that predisposes to sudden cardiac death; co-existing pharmacological therapy; and impaired coronary hemodynamics including coronary blood flow reserve. A number of current investigations are focusing on the possibility that reduced coronary blood flow and flow reserve predispose the hypertensive patient with LVH to premature mortality. Those factors that may participate in potentially lethal ischemia include: reduced basal coronary blood flow and coronary flow reserve; increased coronary vascular resistance and minimal coronary vascular resistance; reduced vasodilating capability of the hypertensive coronary arterioles (e.g., from reduced endothelial synthesis of nitric oxide and other substances); and altered blood viscosity in the coronary microcirculation." @default.
- W33601929 created "2016-06-24" @default.
- W33601929 creator A5021624342 @default.
- W33601929 date "1997-01-01" @default.
- W33601929 modified "2023-09-24" @default.
- W33601929 title "Hypertension, Left Ventricular Hypertrophy, and Coronary Flow Reserve" @default.
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- W33601929 doi "https://doi.org/10.1007/978-1-4615-5385-4_27" @default.
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