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- W336630 endingPage "354" @default.
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- W336630 abstract "This chapter explores the regulation of feeding behavior and energy homeostasis from the perspective of neuroendocrinology. Different techniques have been used to localize Lepr in the brain. The first studies used in situ hybridization to detect the LepR mRNA in rat brain, and found significant expression in parts of the hypothalamus, thalamus, hindbrain and cerebellum. The suppression of food intake by peripheral and central leptin administration was one of the first of its properties to be investigated, and has remained a primary target for investigation ever since. Insulin administered directly into the brain decreases food intake and has effects on glucose homeostasis. Three non-exclusive mechanisms have been proposed to help explain these findings, and each shares some overlap with the way that leptin regulates food intake. Endocrine cells in the stomach, and proximal and distal intestine, are the principal source of peptide hormones that influence food intake and energy metabolism by directly targeting specific neurons in the brain and/or by modulating vagal nerve function." @default.
- W336630 created "2016-06-24" @default.
- W336630 creator A5030003327 @default.
- W336630 date "2012-01-01" @default.
- W336630 modified "2023-09-26" @default.
- W336630 title "Neuroendocrine Regulation of Food Intake" @default.
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