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- W338383485 abstract "The acute respiratory distress syndrome (ARDS) is characterized by progressive hypoxemia, decreased pulmonary compliance, and roentgenographic evidence of diffuse pulmonary infiltrates. The mechanisms that initiate, and then perpetuate the lung inflammation seen in ARDS remain poorly understood. Initially the syndrome was thought to involve only the lungs and to be the result of a single process; activation of the complement cascade. Subsequent investigations have shown that the injury is neither limited to the lungs nor is the pathogenesis simple. Neutrophil sequestration and migration within the lung remain histologic hallmarks of ARDS. It is likely that neutrophil recruitment and subsequent retention are the result of both chemotactic stimuli released within the lungs and activation of neutrophils by circulating mediators. Both complement components and lipopolysaccharide (LPS), a major component of the outer membrane of Gram-negative bacteria, have been implicated as important agents in the induction of ARDS." @default.
- W338383485 created "2016-06-24" @default.
- W338383485 creator A5081203549 @default.
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- W338383485 date "1998-01-01" @default.
- W338383485 modified "2023-09-24" @default.
- W338383485 title "Complement and Endotoxin in Lung Injury" @default.
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- W338383485 doi "https://doi.org/10.1007/978-3-642-60733-2_4" @default.
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