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- W348577146 abstract "Targeted disruption of the murine cytochrome P450 reductase (CPR), the essential electron donor to most microsomal cytochrome P450s, leads to global inactivity of cytochrome P450s in mouse embryos. These mutant embryos die before 10.5 dpc and display multiple defects which include unclosed neural tube, lack of branchial arches and limb buds, an enlarged heart, absence of body turning and shortening of the anteroposterior axis. The vasculature of CPR−/− mutants was severely disrupted with the absence of both major vessels and most capillary beds. Trabeculation in the ventricles and cardiac looping did not occur. Recent reports have established a link between cytochrome P450 catalysed arachidonic acid metabolites and vascular homeostasis in adult mammals. Moreover our data support a novel role for the cytochrome P450 system in the earliest steps of vasculogenesis in mouse post-implantation development." @default.
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- W348577146 date "2002-11-01" @default.
- W348577146 modified "2023-09-27" @default.
- W348577146 title "22 Vasculogenesis defects in cytochrome P450 reductase null mouse embryos." @default.
- W348577146 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/1570964" @default.
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