Matches in SemOpenAlex for { <https://semopenalex.org/work/W36551341> ?p ?o ?g. }
- W36551341 endingPage "589" @default.
- W36551341 startingPage "569" @default.
- W36551341 abstract "ROS including •NO have profound effects on neuronal function. We have come a long way in our understanding of Paul Bert’s observation that high pressures of oxygen cause seizures. Sites sensitive to redox regulation are clearly a factor in both presynaptic transmitter release and the postsynaptic response to neurotransmitters. Transmitter release is decreased by •O−2, H2O2 and •OH as well as •NO and may be further modulated by redox modification of K+channels in the neuronal membrane. The response of the NMDA receptor/channel is highly sensitive to oxidation and reduction and at least one of the redox sites is extracellular. Function of this important receptor may be fined –tuned by alterations in the local redox environment. As a result, overall neuronal excitability in various regions of the CNS may be affected. Unfortunately, the potential for interaction of the reactive molecules and the complexity of the biochemical pathways used by oxyradicals often make interpretation of experimental data difficult. Continued exploration with well–defined “donor” agents and inhibitors will help to resolve the issues of which ROS are neuroactive, how they regulate neuronal function, and when they become neurotoxic." @default.
- W36551341 created "2016-06-24" @default.
- W36551341 creator A5012471774 @default.
- W36551341 creator A5039670204 @default.
- W36551341 date "2006-03-07" @default.
- W36551341 modified "2023-09-23" @default.
- W36551341 title "Reactive Oxygen Species and Neuronal Function" @default.
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