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• W372124429 abstract "Prostacyclin (PGI2), a major product of prostaglandin endoperoxide (PGH2) metabolism in blood vessels, has potent vasodilator and platelet antiaggregatory activity (1). Therefore, modulation of PGI2 synthetase activity is of prime physiological importance in the regulation of blood vessel function. Reducing agents, such as GSH, have been reported to augment prostaglandin production by protecting the cyclooxygenase enzyme system (arachidonic acid → PGH2) from self-catalyzed deactivation (2). However, little evidence exists for the effects of these agents upon augmenting prostacyclin synthetase activity." @default.
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• W372124429 date "1985-01-01" @default.
• W372124429 modified "2023-09-26" @default.
• W372124429 title "Modulation of Coronary Arterial Prostacyclin Synthetase Activity" @default.
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• W372124429 doi "https://doi.org/10.1007/978-1-4613-2589-5_29" @default.
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