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- W37304025 abstract "Prostaglandins (PGs) and leukotrienes (LTs) have long been known to be important endogenous mediators of inflammation (for reviews see refs. 1,2). The pathways for PG formation were elucidated first in the early 1960s by Bergström et al. (3), and by van Dorp et al. (4), and those for the LTs in 1979–1980 (5,6). Their biological actions were assessed by classical pharmacological experiments involving bioassays and addition of cyclooxygenese (COX) or lipoxygenase (LO) inhibitors or receptor antagonists. The early discovery of nonsteroidal antiinflammatory drugs (NSAID), like aspirin and indomethacin which specifically inhibit COX enzyme activity (7,8), greatly simplified the task of ascertaining the role of PGs in inflammation. Progress in defining specific inhibitors and receptor antagonists for the LT pathway was relatively slow in comparison." @default.
- W37304025 created "2016-06-24" @default.
- W37304025 creator A5032705392 @default.
- W37304025 date "1999-01-01" @default.
- W37304025 modified "2023-10-17" @default.
- W37304025 title "Lipid-Mediator-Deficient Mice in Models of Inflammation" @default.
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- W37304025 doi "https://doi.org/10.1007/978-1-59259-253-1_5" @default.
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