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- W39164435 endingPage "4288" @default.
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- W39164435 abstract "Triple-helix DNA structures can form endogenously at mirror repeat polypurine/polypyrimidine sequences or by introduction of triplex-forming oligonucleotides (TFOs). Recent evidence suggests that triple helices are sources of genetic instability, and are subject to increased rates of mutagenesis and recruitment of repair factors. Indeed, observations using TFOs suggest that triple helices provoke a variety of biological processes which can be harnessed to modulate gene expression and induce heritable changes in targeted genes. This review surveys the biological applications of TFOs, with particular attention to their recombinogenic and mutagenic potential, and summarizes available evidence for the mechanism of triplex and triplex-associated repair." @default.
- W39164435 created "2016-06-24" @default.
- W39164435 creator A5081896149 @default.
- W39164435 date "2007-01-01" @default.
- W39164435 modified "2023-09-23" @default.
- W39164435 title "Repair and recombination induced by triple helix DNA" @default.
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- W39164435 doi "https://doi.org/10.2741/2388" @default.
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