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- W40889683 abstract "There is a historical causative association between arthritis and infection. This has now been firmly established as an aetiological relationship following advances in the fields of microbiology, immunogenetics and clinical rhematology. Astute observation and belief in an infective cause for arthritis has provided rheumatologists with many of the current anti-arthritic drugs. At the end of the last century, gold was shown to inhibit growth of tubercle bacilli and Forestier, assuming tuberculosis and rheumatoid shared a common aetiology, showed it to be effective for arthritis1. Gold also inhibits mycoplasma, which have also been implicated in causing rheumatoid arthritis2. Sulphasalazine was developed in the belief that rheumatoid arthritis was infective in origin3. Following initially disappointing trials, it has now been shown to be effective in both ankylosing spondylitis4 and rheumatoid arthritis5. In rheumatoid arthritis, it is the bacteriostatic sulphapyridine that is the active moiety6. The sulphones, dapsone7 and sulphamethoxazole8 are also effective in rheumatoid arthritis. There are many other drugs, i.e. chloroquine, levamisole, metronidazole, clotrimazole, rifampicin, derived for infective conditions, which have been used with variable success in the treatment of rheumatoid arthritis." @default.
- W40889683 created "2016-06-24" @default.
- W40889683 creator A5052406633 @default.
- W40889683 creator A5088856926 @default.
- W40889683 date "1989-01-01" @default.
- W40889683 modified "2023-09-25" @default.
- W40889683 title "The Relationship Between Bacteria, Related Organisms and Chronic Arthritis" @default.
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