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• W4168069 abstract "In chronic renal failure patients, the most frequently observed deviation from normal is a reduction of serum tri-iodothyronine (T3), both total and free tri-iodothyronine (TT3 and FT3) concentrations are reduced. Serum total thyroxine (TT4) is also reduced but in a more minor magnitude and with a lesser frequency. Serum-free T4 index (FT4I) is likewise reduced. Serum free T4 ranged from subnormal to elevated levels depending on the assay method. Thyroxine-binding globulin is generally normal or only mildly depressed. Serum total reverse T3 (rT3), which is never measured in clinical settings but assessed in research studies, is either normal or elevated. Serum free rT3 is high. Serum thyrotropin or thyroid-stimulating hormone (TSH) is not elevated, but it is uniformly normal. The low T3 is unequivocally a result of reduced production; the T4 to T3 conversion rate is markedly impaired at extrathyroidal sites. However, its metabolic clearance and degradation rates are actually reduced. The pathogenesis of the low T4 is more complex. Both T4 degradation and production rates, the latter equal to thyroidal T4 secretion rate, are normal. The major contributing factor is a defect in T4 binding to circulating protein carriers. A concomitant defect in central hypothalamic thyrotropin-releasing hormone secretion cannot be excluded, especially in those patients whose both T4 and serum thyrotropin concentrations are severely reduced." @default.
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• W4168069 date "2009-01-01" @default.
• W4168069 modified "2023-10-16" @default.
• W4168069 title "Thyroid Status in Chronic Renal Failure Patients – A Non-Thyroidal Illness Syndrome" @default.
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• W4168069 doi "https://doi.org/10.1016/b978-0-12-373870-7.00028-4" @default.
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