Matches in SemOpenAlex for { <https://semopenalex.org/work/W4200073201> ?p ?o ?g. }
- W4200073201 endingPage "168" @default.
- W4200073201 startingPage "155" @default.
- W4200073201 abstract "Patients affected by endometriosis can experience a variety of pain symptoms, such as dysmenorrhea, chronic pelvic pain, dyspareunia, and dyschezia. Similarly to eutopic endometrium, endometriotic lesions are influenced by endocrine environment and subjected to cyclic modifications during the menstrual cycle. Retrograde menstruation of endometrial tissue and intermenstrual bleeding of endometriotic lesions may directly contribute to inflammation with subsequent activation of innate immune cells and mediators within implants. The secretion of pronociceptive and proinflammatory mediators contributes to the generation of nociceptive signals. Vice versa, nerve fibers, especially abundant in deep endometriotic lesions, also have an active role in the mechanism of inflammatory pain by secreting proinflammatory neuropeptides. In patients with a long-standing history of endometriosis, central neural processing can be altered; therefore, initially peripherally induced central sensitization becomes independent of peripheral sensitization. The growing understanding of the mechanisms that are responsible for the development of pain related to endometriosis is allowing the investigation of new medical opportunities." @default.
- W4200073201 created "2021-12-31" @default.
- W4200073201 creator A5012645361 @default.
- W4200073201 creator A5046855515 @default.
- W4200073201 creator A5064812826 @default.
- W4200073201 creator A5090663007 @default.
- W4200073201 date "2022-01-01" @default.
- W4200073201 modified "2023-09-25" @default.
- W4200073201 title "Pain" @default.
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