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- W4200099132 abstract "Hyperkalemia is uncommon in the presence of normal renal function. It can result from excessive intake, inadequate excretion, or shifting of potassium from cells into plasma. Translocation of potassium out of cells can cause mild to moderate hyperkalemia. This is seen with metabolic acidosis as a result of excess hydrogen ions being buffered in cells and potassium being exchanged to maintain electroneutrality. The clinical signs associated with hyperkalemia are usually a result of changes in the resting membrane potential of the cells. The increased extracellular potassium concentration makes the resting membrane potential less negative. This change brings the resting membrane potential and threshold potentials closer together, making the membrane more excitable. Sodium bicarbonate also will help lower serum potassium levels by causing the potassium to shift into the cells, in exchange for the hydrogen ion, which moves into the extracellular space." @default.
- W4200099132 created "2021-12-31" @default.
- W4200099132 creator A5087307006 @default.
- W4200099132 date "2021-12-20" @default.
- W4200099132 modified "2023-09-23" @default.
- W4200099132 title "Hyperkalemia" @default.
- W4200099132 doi "https://doi.org/10.1201/9781315140629-5" @default.
- W4200099132 hasPublicationYear "2021" @default.
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