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- W4200407601 abstract "Ulcerative colitis (UC), an etiologically complicated and relapsing gastrointestinal disease, is characterized by the damage of mucosal epithelium and destruction of the intestinal homeostasis, which has caused a huge social and economic burden on the health system all over the world. Its pathogenesis is multifactorial, including environmental factors, genetic susceptibility, epithelial barrier defect, symbiotic flora imbalance, and dysregulated immune response. Thus far, although immune cells have become the focus of most research, it is increasingly clear that intestinal epithelial cells play an important role in the pathogenesis and progression of UC. Notably, apoptosis is a vital catabolic process in cells, which is crucial to maintain the stability of intestinal environment and regulate intestinal ecology. In this review, the mechanism of apoptosis induced by reactive oxygen species and endoplasmic reticulum stress, as well as excessive apoptosis in intestinal epithelial dysfunction and gut microbiology imbalance are systematically and comprehensively summarized. Further understanding the role of apoptosis in the pathogenesis of UC may provide a novel strategy for its therapy in clinical practices and the development of new drugs.Recently, the prevalence of ulcerative colitis (UC) has increased, but the pathogenesis of UC remains poorly understood. A better understanding of the role of apoptosis in the pathogenesis of UC may provide a promising prospect for UC treatment." @default.
- W4200407601 created "2021-12-31" @default.
- W4200407601 creator A5003181280 @default.
- W4200407601 creator A5031980964 @default.
- W4200407601 creator A5040817687 @default.
- W4200407601 creator A5042290112 @default.
- W4200407601 creator A5064313538 @default.
- W4200407601 date "2021-12-06" @default.
- W4200407601 modified "2023-10-16" @default.
- W4200407601 title "Excessive Apoptosis in Ulcerative Colitis: Crosstalk Between Apoptosis, ROS, ER Stress, and Intestinal Homeostasis" @default.
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