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- W4200418061 endingPage "103016" @default.
- W4200418061 startingPage "103016" @default.
- W4200418061 abstract "Systemic autoimmune and inflammatory diseases have a complex and only partially known pathophysiology with various abnormalities involving all the components of the immune system. Among these components, antibodies, and especially autoantibodies are key elements contributing to autoimmunity. The interaction of antibody fragment crystallisable (Fc) and several distinct receptors, namely Fc receptors (FcRs), have gained much attention during the recent years, with possible major therapeutic perspectives for the future. The aim of this review is to comprehensively describe the known roles for FcRs (activating and inhibitory FcγRs, neonatal FcR [FcRn], FcαRI, FcεRs, Ro52/tripartite motif containing 21 [Ro52/TRIM21], FcδR, and the novel Fc receptor-like [FcRL] family) in systemic autoimmune and inflammatory disorders, namely rheumatoid arthritis, Sjögren's syndrome, systemic lupus erythematosus, systemic sclerosis, idiopathic inflammatory myopathies, mixed connective tissue disease, Crohn's disease, ulcerative colitis, immunoglobulin (Ig) A vasculitis, Behçet's disease, Kawasaki disease, IgG4-related disease, immune thrombocytopenia, autoimmune hemolytic anemia, antiphospholipid syndrome and heparin-induced thrombocytopenia." @default.
- W4200418061 created "2021-12-31" @default.
- W4200418061 creator A5006550508 @default.
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- W4200418061 creator A5083197907 @default.
- W4200418061 creator A5090766654 @default.
- W4200418061 date "2022-03-01" @default.
- W4200418061 modified "2023-10-13" @default.
- W4200418061 title "Fc receptors gone wrong: A comprehensive review of their roles in autoimmune and inflammatory diseases" @default.
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