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- W4200423719 endingPage "14" @default.
- W4200423719 startingPage "14" @default.
- W4200423719 abstract "Its increasing incidence has led stroke to be the second leading cause of death worldwide. Despite significant advances in recanalization strategies, patients are still at risk for ischemia/reperfusion injuries in this pathophysiology, in which neuroinflammation is significantly involved. Research has shown that in the acute phase, neuroinflammatory cascades lead to apoptosis, disruption of the blood-brain barrier, cerebral edema, and hemorrhagic transformation, while in later stages, these pathways support tissue repair and functional recovery. The present review discusses the various cell types and the mechanisms through which neuroinflammation contributes to parenchymal injury and tissue repair, as well as therapeutic attempts made in vitro, in animal experiments, and in clinical trials which target neuroinflammation, highlighting future therapeutic perspectives." @default.
- W4200423719 created "2021-12-31" @default.
- W4200423719 creator A5020808759 @default.
- W4200423719 creator A5053100396 @default.
- W4200423719 date "2021-12-21" @default.
- W4200423719 modified "2023-10-18" @default.
- W4200423719 title "Neuroinflammation in Cerebral Ischemia and Ischemia/Reperfusion Injuries: From Pathophysiology to Therapeutic Strategies" @default.
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