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- W4200596985 abstract "The 2019 coronavirus disease (COVID-19) is the disease caused by SARS-CoV-2 infection. Although this infection has been shown to affect the respiratory system, a high incidence of thrombotic events has been observed in severe cases of COVID-19 and in a significant portion of COVID-19 nonsurvivors. Although prior literature has reported on both the coagulopathy and hypercoagulability of COVID-19, the specifics of coagulation have not been fully investigated. Observations of microthrombosis in patients with COVID-19 have brought attention to potential inflammatory endothelial injury. Von Willebrand factor (VWF) and its protease, A disintegrin and metalloproteinase with a thrombospondin type 1 motif, member 13 (ADAMTS13), play an important homeostatic role in responding to endothelial injury. This report provides an overview of the literature investigating the role the VWF/ADAMTS13 axis may have in COVID-19 thrombotic events and suggests potential therapeutic strategies to prevent the progression of coagulopathy in patients with COVID-19." @default.
- W4200596985 created "2021-12-31" @default.
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- W4200596985 date "2022-01-01" @default.
- W4200596985 modified "2023-10-10" @default.
- W4200596985 title "Contribution of the von Willebrand factor/ADAMTS13 imbalance to COVID-19 coagulopathy" @default.
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- W4200596985 doi "https://doi.org/10.1152/ajpheart.00204.2021" @default.
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