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- W4200609241 endingPage "13154" @default.
- W4200609241 startingPage "13154" @default.
- W4200609241 abstract "The identification of novel strategies to control Helicobacter pylori (Hp)-associated chronic inflammation is, at present, a considerable challenge. Here, we attempt to combat this issue by modulating the innate immune response, targeting formyl peptide receptors (FPRs), G-protein coupled receptors that play key roles in both the regulation and the resolution of the innate inflammatory response. Specifically, we investigated, in vitro, whether Caulerpin-a bis-indole alkaloid isolated from algae of the genus Caulerpa-could act as a molecular antagonist scaffold of FPRs. We showed that Caulerpin significantly reduces the immune response against Hp culture filtrate, by reverting the FPR2-related signaling cascade and thus counteracting the inflammatory reaction triggered by Hp peptide Hp(2-20). Our study suggests Caulerpin to be a promising therapeutic or adjuvant agent for the attenuation of inflammation triggered by Hp infection, as well as its related adverse clinical outcomes." @default.
- W4200609241 created "2021-12-31" @default.
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- W4200609241 date "2021-12-05" @default.
- W4200609241 modified "2023-09-30" @default.
- W4200609241 title "Caulerpin Mitigates Helicobacter pylori-Induced Inflammation via Formyl Peptide Receptors" @default.
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- W4200609241 doi "https://doi.org/10.3390/ijms222313154" @default.
- W4200609241 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/34884957" @default.