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- W4205493669 abstract "X-linked hypophosphatemia (XLH), the most common form of hereditary hypophosphatemic rickets, is caused by inactivating mutations of the phosphate-regulating endopeptidase gene (PHEX). XLH is mainly characterized by short stature, bone deformities and rickets, while in hypophosphatemia, normal or low vitamin D levels and low renal phosphate reabsorption are the principal biochemical aspects. The cause of growth impairment in patients with XLH is not completely understood yet, thus making the study of the growth plate (GP) alterations necessary. New treatment strategies targeting FGF23 have shown promising results in normalizing the growth velocity and improving the skeletal effects of XLH patients. However, further studies are necessary to evaluate how this treatment affects the GP as well as its long-term effects and the impact on adult height." @default.
- W4205493669 created "2022-01-25" @default.
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- W4205493669 date "2022-01-15" @default.
- W4205493669 modified "2023-10-18" @default.
- W4205493669 title "Cellular and Molecular Alterations Underlying Abnormal Bone Growth in X-Linked Hypophosphatemia" @default.
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- W4205493669 doi "https://doi.org/10.3390/ijms23020934" @default.
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