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• W4206464080 abstract "Ischemia-reperfusion (I/R) injury is a manifestation of tissue or organ damage that is followed by ischemia and exacerbated by the return of blood flow to a previously damaged tissue or organ. The intestines are one of the most sensitive tissues and organs to I/R injury. Moreover, the adverse consequences of intestinal I/R (II/R) injury are not limited to the intestine itself and can also lead to damage of the distant tissues and organs. The mechanism of II/R is extremely complex and oxidative stress is the key link in the pathogenesis of II/R injury. This study summarizes the roles of oxidative stress and its signaling pathways involved in II/R. The signaling pathways that mitigate II/R injury include the nuclear factor erythroid-related factor 2 (Nrf2)-mediated signaling pathway, Wnt/β-catenin pathway, and phosphatidylinositol kinase 3 (PI3K)/Akt pathway; those that aggravate II/R injury include the Janus kinase/signal transducer and activator of transcription (JAK/STAT) pathway, Toll-like receptor (TLR) receptor-mediated signaling pathway, protein kinase CβII (PKCβII)/p66shc pathway, and microRNA (miRNA)/p66shc pathway; the effect of miRNA on related pathways and mitochondrial DNA translocation. The aforementioned pathways provide new ideas for further exploring the occurrence and development of II/R and more effective treatments for II/R injury." @default.
• W4206464080 created "2022-01-26" @default.
• W4206464080 creator A5003432174 @default.
• W4206464080 creator A5013878126 @default.
• W4206464080 creator A5067321308 @default.
• W4206464080 date "2022-01-14" @default.
• W4206464080 modified "2023-10-17" @default.
• W4206464080 title "Oxidative Stress in Intestinal Ischemia-Reperfusion" @default.
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• W4206464080 doi "https://doi.org/10.3389/fmed.2021.750731" @default.
• W4206464080 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/35096858" @default.
• W4206464080 hasPublicationYear "2022" @default.
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