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- W4210480084 abstract "Virtually any stressor that alters the cellular homeostatic state may result in an inflammatory response. As a critical component of innate immunity, inflammasomes play a prominent role in the inflammatory response. The information on inflammasome biology is rapidly growing, thus creating the need for structuring it into a model that can help visualize and enhance the understanding of underlying biological processes. Causal biological network (CBN) models provide predictive power for novel disease mechanisms and treatment outcomes. We assembled the available literature information on inflammasome activation into the CBN model and scored it with publicly available transcriptomic datasets that address viral infection of the lungs, osteo- and rheumatoid arthritis, psoriasis, and aging. The scoring inferred pathway activation leading to NLRP3 inflammasome activation in these diverse conditions, demonstrating that the CBN model provides a platform for interpreting transcriptomic data in the context of inflammasome activation." @default.
- W4210480084 created "2022-02-08" @default.
- W4210480084 creator A5011233293 @default.
- W4210480084 creator A5053633773 @default.
- W4210480084 creator A5089030305 @default.
- W4210480084 date "2022-01-27" @default.
- W4210480084 modified "2023-09-25" @default.
- W4210480084 title "Causal Biological Network Model for Inflammasome Signaling Applied for Interpreting Transcriptomic Changes in Various Inflammatory States" @default.
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- W4210480084 doi "https://doi.org/10.1155/2022/4071472" @default.
- W4210480084 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/35126992" @default.
- W4210480084 hasPublicationYear "2022" @default.
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