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- W4211124197 endingPage "454" @default.
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- W4211124197 abstract "The pathophysiology of Parkinson's disease motor symptoms and the development of involuntary movements, dyskinesia, following long-term levodopa therapy involve a number of neurotransmitters and neuromodulators within the basal ganglia circuitry that provide novel therapeutic targets. In this respect, acetylcholine, adenosine, glutamate, serotonin, adrenergic, cannabinoid, opioid, and histaminergic systems have been most closely studied. Several drugs that target these receptors are in clinical use, with many more in clinical development. To date, lack of a complete understanding of the neural mechanisms underlying the action of these neurotransmitters, lack of clinically available subtype selective agents, and tolerability have slowed translation into daily clinical practice. However, the future clinical usefulness of these drugs still lies in a potential ability to enhance the anti-parkinsonian action of levodopa and to reduce motor and non-motor complications." @default.
- W4211124197 created "2022-02-13" @default.
- W4211124197 creator A5018228116 @default.
- W4211124197 creator A5048688457 @default.
- W4211124197 date "2011-06-27" @default.
- W4211124197 modified "2023-09-30" @default.
- W4211124197 title "Non‐Dopaminergic Approaches to the Treatment of Parkinson's Disease" @default.
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