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- W4211133027 endingPage "500" @default.
- W4211133027 startingPage "487" @default.
- W4211133027 abstract "Dying mammalian cells emit numerous signals that interact with the host to dictate the immunological correlates of cellular stress and death. In the absence of reactive antigenic determinants (which is generally the case for healthy cells), such signals may drive inflammation but cannot engage adaptive immunity. Conversely, when cells exhibit sufficient antigenicity, as in the case of infected or malignant cells, their death can culminate with adaptive immune responses that are executed by cytotoxic T lymphocytes and elicit immunological memory. Suggesting a key role for immunogenic cell death (ICD) in immunosurveillance, both pathogens and cancer cells evolved strategies to prevent the recognition of cell death as immunogenic. Intriguingly, normal cells succumbing to conditions that promote the formation of post-translational neoantigens (for example, oxidative stress) can also drive at least some degree of antigen-specific immunity, pointing to a novel implication of ICD in the etiology of non-infectious, non-malignant disorders linked to autoreactivity." @default.
- W4211133027 created "2022-02-13" @default.
- W4211133027 creator A5060740655 @default.
- W4211133027 creator A5071827163 @default.
- W4211133027 creator A5074612242 @default.
- W4211133027 creator A5075296559 @default.
- W4211133027 date "2022-02-10" @default.
- W4211133027 modified "2023-10-13" @default.
- W4211133027 title "Immunogenic cell stress and death" @default.
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